Branched-chain amino acid catabolic defect in vascular smooth muscle cells drives thoracic aortic dissection via mTOR hyperactivation.
Study designOther primary literature
SubjectHuman & animal
Abstract
cc by 以外(cc by-nc / cc by-nc-nd / NONE 等)は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。
MeSH
DOI 10.1016/j.freeradbiomed.2023.11.002
PMID 37956909
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