Autophagy activated by GR/miR-421-3p/mTOR pathway as a compensatory mechanism participates in chondrodysplasia induced by prenatal caffeine exposure in male fetal rats.

Toxicology letters2024Han H, Shi H, Jiang L, et al.

Study designOther primary literature

SubjectAnimal

Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

AnimalsAutophagyCaffeineChondrocytesFemaleMaleMicroRNAsPregnancyPrenatal Exposure Delayed EffectsRatsRats, Sprague-DawleySignal TransductionTOR Serine-Threonine Kinases

DOI 10.1016/j.toxlet.2024.05.010

PMID 38759937

View source →