Dysfunctional BCAA degradation triggers neuronal damage through disrupted AMPK-mitochondrial axis due to enhanced PP2Ac interaction.

Communications biology2025Wu SC, Chen YJ, Su SH, et al.

Study designOther primary literature

SubjectAnimal

Abstract

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MeSH

AMP-Activated Protein KinasesAmino Acids, Branched-ChainAnimalsAutophagyDrosophila ProteinsDrosophila melanogasterMitochondriaNeuronsProtein Phosphatase 2Reactive Oxygen Species

DOI 10.1038/s42003-025-07457-6

PMID 39838082

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