Role of endothelial nitric oxide synthase-derived nitric oxide in activation and dysfunction of cerebrovascular endothelial cells during early onsets of sepsis.

American journal of physiology. Heart and circulatory physiology2008Handa O, Stephen J, Cepinskas G

Study designOther primary literature

SubjectAnimal

Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

AnimalsAntioxidantsBrainCell Membrane PermeabilityCells, CulturedDisease Models, AnimalEndothelium, VascularEnzyme InhibitorsMembrane ProteinsMiceMice, Inbred C57BLMice, KnockoutNitric OxideNitric Oxide Synthase Type IINitric Oxide Synthase Type IIIOccludinOxidative StressReactive Oxygen SpeciesSepsisTime Factors

DOI 10.1152/ajpheart.00476.2008

PMID 18723768

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