Inhibiting c-Jun N-terminal kinase partially attenuates caffeine-dependent cell death without alleviating the caffeine-induced reduction in mitochondrial respiration in C2C12 skeletal myotubes.

Biochemical and biophysical research communications2016Downs RM, Hughes MA, Kinsey ST, et al.

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MeSH

AnimalsAnthracenesCaffeineCaspase 3Cell DeathCell LineDantroleneDose-Response Relationship, DrugEndoplasmic Reticulum StressJNK Mitogen-Activated Protein KinasesMiceMitochondriaMuscle Fibers, SkeletalProtein Kinase Inhibitors

DOI 10.1016/j.bbrc.2016.10.008

PMID 27717822

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